New PDF release: Advanced Imaging In Coronary Artery Disease: PET, SPECT,

By Allard C. van der Wal, Anton E. Becker (auth.), Ernst E. Van Der Wall, Paul K. Blanksma, Menco G. Niemeyer, Willem Vaalburg, Harry J. G. M. Crijns (eds.)

ISBN-10: 9400708661

ISBN-13: 9789400708662

ISBN-10: 9401037469

ISBN-13: 9789401037464

In Advanced Imaging in Coronary Artery Disease, the position of a number of imaging strategies in diagnosing atherosclerosis, evaluate of myocardial ischemia, myocardial viability, and middle failure are extensively mentioned. the problems derived from cardiac puppy are awarded on the subject of the traditional innovations, similar to echocardiography, SPECT and MRI. moreover, more moderen imaging options akin to intracoronary ultrasound, electron beam computed tomography, and Raman spectroscopy are given huge cognizance. the consequences of drug therapy, akin to anti-ischemic and lipid-lowering medicines, also are evaluated.
This e-book will support medical cardiologists, nuclear medication physicians, fellows in cardiology and nuclear drugs, radiochemists, easy study fellows, and technicians, in realizing the hot advances in scientific cardiac PET.

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Extra info for Advanced Imaging In Coronary Artery Disease: PET, SPECT, MRI, IVUS, EBCT

Sample text

Smoking did not influence the composition of the plaques with respect to features of vulnerability, but appeared to be highly thrombogenic. Moreover, 69% of deep plaque ruptures reaching into a lipid core were found in man, whereas in a previous study of the same group 42, 69% of superficial erosions was reported in women {in plaques composed of SMC and matrix proteins rather than lipids 11 and macrophages), indicating sex related differences in the type of rupture, and probably also plaque composition.

Moreno PR, Falk E, Palacios IF, Newell JB, Fuster V, Fallon JT. Macrophage infiltration in acute coronary syndromes. Implications for plaque rupture. Circulation, 1994;90:775-8. Brown DL, Hibbs MS, Keaney M, Loushin C, Isner JM. Identification of 92 kD gelatinase in human coronary atherosclerotic lesions: Association of active enzyme synthesis with unstable angina. Circulation 1995;91 :2125-31. Haberbosch W, Bohle RM, Franke FE, et al. The expression of angiotensin-I converting enzyme in human atherosclerotic plaques is not related to the deletion/inserton polymorphism but to the risk of restenosis after coronary interventions.

19 Therefore it is possible to evaluate stent expansion and tissue apposition accurately (figure 1). Figure 1. IVUS recording after stent implantation, showing adequate stent expansion and optimal apposition of the struts (See also Colour Plates, p. 337) 19 The aforementioned characteristics of stent deployment are related to plaque composition and compliance. Fibro-calcified lesions are less easily deformed. 20 These findings resulted in higher inflation pressures for stent delivery and a subsequent reduction of the rate of suboptimal deployment to 30-40%.

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Advanced Imaging In Coronary Artery Disease: PET, SPECT, MRI, IVUS, EBCT by Allard C. van der Wal, Anton E. Becker (auth.), Ernst E. Van Der Wall, Paul K. Blanksma, Menco G. Niemeyer, Willem Vaalburg, Harry J. G. M. Crijns (eds.)


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